Download Dental Caries: The Disease and Its Clinical Management by Ole Fejerskov, Bente Nyvad, Edwina Kidd PDF

By Ole Fejerskov, Bente Nyvad, Edwina Kidd

The second one variation of Dental Caries: the sickness and its medical administration builds at the luck of the distinguished first version to offer an unequalled source on cariology. The medical thrust of the 1st variation is widened and bolstered to incorporate assurance of the illness in all its type, from eruption of the 1st basic enamel to the time-honored sorts of the disorder in older sufferers. The centrality of caries keep an eye on and administration to the dental well-being of all populations is extra emphasised, because the booklet is going past the winning therapy of carious lesions to illustrate the long term results of the non-operative and healing concepts hired.

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Extra info for Dental Caries: The Disease and Its Clinical Management

Example text

These two latter terms are synonyms, but in this textbook we will use the term recurrent caries throughout. Recurrent caries is simply a lesion developing at a tooth surface adjacent to a filling. As such, its etiology is similar to that of primary caries. Residual caries, as the term implies, is demineralized tissue that has been left behind before a filling is placed. An important classification is whether a lesion is cavitated or non-cavitated, as it impinges directly on the management of the lesion (Chapter 4).

3 Upper deciduous canine from a 5-year-old with an active, cavitated lesion along the gingival margin. On probing it would be soft, but there is no reason to probe such a lesion unless you wish to provoke a pain reaction! 2 Inactive or arrested caries lesions on buccal surfaces of upper central incisor teeth in a 5-year-old child. Note that the shape of the lesions indicates where the gingival margin was located at the time when these lesions developed. The oral hygiene is now improved and the surfaces of these noncavitated opaque lesions are smooth and shiny.

33). This zone may vary from 5 to 100 μm in width and is located corresponding to that part of the lesion with a pore volume of slightly more than 1% when examined in dry air. Detailed microdensitometry studies of microradiograms have shown that there is a slight loss of mineral in this zone. The explanation for the translucent appearance of this zone with the enamel structures being less evident appears to be that initial dissolution of the enamel mainly occurs along the gaps between rod and the interrod enamel in the tissue.

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