By Richard C.K. Jordan, Michael A.O. Lewis
A finished consultant to analysis and remedy of oral problems;superb color illustrations built-in all through - combining the benefits of an atlas with a brief text/reference;symptoms-based approach;one situation in keeping with web page or web page spread.
content material: PREFACE; ACKNOWLEDGEMENTS; ABBREVIATIONS; 1 advent; 2 ULCERATION; three BLISTERS; four WHITE PATCHES; five ERYTHEMA; 6 SWELLING; 7 PIGMENTATION (INCLUDING BLEEDING); eight OROFACIAL ache (INCLUDING SENSORY AND MOTOR DISTURBANCE); nine DRY MOUTH, extra SALIVATION, lined TONGUE, HALITOSIS, and adjusted flavor; INDEX.
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Additional resources for A Colour Handbook of Oral Medicine
Example text
Both eyes were similarly affected. 53 54 BLISTERS Pemphigus ETIOLOGY AND PATHOGENESIS Pemphigus comprises a group of auto-immune, vesiculobullous disorders characterized by involvement of the skin, mouth, and other mucous membrane sites. Forms of pemphigus may be differentiated on the basis of the level of intra-epithelial involvement. Pemphigus vulgaris and pemphigus vegetans, the two forms that may produce oral lesions, affect the full width of the epithelium, while pemphigus foliaceous and pemphigus erythematosus occur in the upper prickle cell layer/spinous layer.
The mucosa of the hard palate is the site most frequently involved (82, 83) but other areas such as the lower buccal sulcus or gingival margins (84) can also be affected. It can be difficult to determine whether the lesion(s) were precipitated by trauma in these patients or whether they chronically shed HSV in their saliva which subsequently colonizes traumatized mucosa. DIAGNOSIS The clinical appearance is usually diagnostic. Confirmation of the presence of HSV can be made by isolation from a swab in tissue culture or the use of immunofluorescence on a smear of a recent lesion.
Patients should be followed up for at least 2 years and serologic examinations repeated over this period. SYPHILIS 57 57 Ulcerated nodular lesion of primary syphilis. 58 58 Ulceration of secondary syphilis. 59 59 Notch deformity of the incisors due to congenital syphilis (Hutchinson’s incisors). 60 60 Hypoplastic deformity of the first molar due to congenital syphilis (Moon’s molar, mulberry molar). 33 34 ULCERATION Acute necrotizing ulcerative gingivitis ETIOLOGY AND PATHOGENESIS The etiology of acute necrotizing ulcerative gingivitis (ANUG) is not fully understood but strictly anerobic bacteria, in particular spirochetes and Fusobacterium species, are likely to be involved since high numbers of these microorganisms can be demonstrated in lesions.